Skeletal muscle pyruvate dehydrogenase activity during maximal exercise in humans. Putman, C. T., N. L. Jones, L. C. Lands, T. M. Bragg, M. G. Hollidge -Horvat, and G. J. F. Heigenhauser. Department of Medicine, McMaster University Medical Centre, Hamilton, Ontario, CANADA L8N 3Z5
APStracts 2:0081E, 1995.
The regulation of the active form of pyruvate dehydrogenase (PDHa) and related metabolic events were examined in human skeletal muscle during repeated bouts of maximum exercise. Seven subjects completed 3 consecutive 30 second bouts of maximum isokinetic cycling, separated by 4 min of recovery. Biopsies of the vastus lateralis were taken before and immediately after each bout. PDHa increased from 0.45+/ -0.15 to 2.96+/-0.38; from 1.10+/-0.11 to 2.91+/-0.11 and from 1.28+/ -0.18 to 2.82+/-0.32 mmol.min-1.kg ww-1 during bouts 1, 2 and 3 respectively. Glycolytic flux was 13 fold greater than PDHa in bouts 1 and 2, and 4 fold greater during bout 3. This discrepancy between the rate of pyruvate production and oxidation resulted in substantial lactate accumulation to 89.5+/-11.6 in bout 1, 130.8+/-13.8 in bout 2 and 106.6+/-10.1 mmol.kg dw-1 in bout 3. These events coincided with an increase in the mitochondrial oxidation state, as reflected in a fall in the mitochondrial NADH-to-NAD ratio, indicating that muscle lactate production during exercise was not an O2 dependent process in our subjects. During exercise the primary factor regulating PDHa transformation was probably intracellular Ca2+. In contrast, the primary regulatory factors causing greater PDHa during recovery were lower ATP-to-ADP and NADH-to-NAD ratios, and increased concentrations of pyruvate and H+. Greater PDHa during recovery facilitated continued oxidation of the lactate load between exercise bouts.

Received 23 February 1994; accepted in final form 12 April 1995.
APS Manuscript Number E86-5.
Article publication pending Am. J. Physiol. (Endocrinol. Metab.).
ISSN 1080-4757 Copyright 1995 The American Physiological Society.
Published in APStracts on 25 April 1995.