Regulation of membrane potential and diameter by voltage-dependent k+ channels in rabbit myogenic cerebral arteries. Knot, Harm J., and Mark T. Nelson. Smooth Muscle Ion Channel Group, Department of Pharmacology, 55A South Park Drive, The University of Vermont, Colchester, Vermont 05446-2500
APStracts 2:0015H, 1995.
The hypothesis that voltage-dependent potassium channels are involved in the regulation of arterial smooth muscle membrane potential and blood vessel diameter was tested by examining the effects of inhibitors (4-aminopyridine (4-AP) and 3,4-diaminopyridine (3,4-DAP)) of voltage-dependent K+ channels on the membrane potential and diameter of pressurized, small (100-300 micron diameters) cerebral arteries from rabbit. In response to graded elevations in transmural pressure (20-100 mm Hg), the membrane potential of smooth muscle cells in these arteries depolarized and the arteries constricted. 4 -AP (1 mM) and 3,4-DAP (1 mM) depolarized cerebral arteries by 19 mV and 21 mV, respectively, when subjected to a transmural pressure of 80 mm Hg. 3-Aminopyridine (3-AP)(1 mM) which is a relatively poor inhibitor of voltage-dependent K+ channels depolarized smooth muscle cells in the arteries by 1 mV. 4-AP and 3,4-DAP constricted pressurized (to 80 mm Hg) cerebral arteries. 3-AP had little effect on arterial diameter. 4-AP increased the arterial constriction to transmural pressure over a wide range of pressures (40-90 mm Hg). The effects of 4-AP and 3,4-DAP on membrane potential and diameter were not prevented by inhibitors of calcium channels, calcium-activated K+ channels, ATP-sensitive K+ channels, inward rectifier K+ channels, or by blockers of adrenergic, serotonergic, muscarinic and histaminergic receptors, or by removal of the endothelium. These results suggest that voltage-dependent potassium channels are involved in the regulation of membrane potential and response of small cerebral arteries to changes in intravascular pressure.

Received 15 September 1994; accepted in final form 26 January
1995.
APS Manuscript Number H828-4.
Article publication pending Am. J. Physiol. (Heart Circ. Physiology).
ISSN 1080-4757 Copyright 1995 The American Physiological Society.
Published in APStracts on 24 February 1995.