TWO TYPES OF NETWORK OSCILLATIONS IN NEOCORTEX MEDIATED BY DISTINCT GLUTAMATE RECEPTOR SUBTYPES AND NEURONAL POPULATIONS. Flint, Alexander C. Alexander C. and Barry W. Connors. Department of Neuroscience, Brown University, Providence, RI 02912.
APStracts 2:0332N, 1995.
SUMMARY AND CONCLUSIONS
1. Two distinct forms of spontaneous synchronous oscillations were investigated with field potential recordings in slices of rat somatosensory cortex in vitro . 2. The first type of synchronous oscillation was activated by low extracellular [Mg 2+ ], and had dominant frequencies of 8-12 Hz. It was reversibly abolished by the NMDA receptor antagonist d-2-amino-5- phosphonovaleric acid (AP-5), and was relatively unaffected by the non-NMDA receptor antagonist 6,7-dinitroquinoxaline-2,3-dione (DNQX). The duration of oscillatory events was increased by blocking GABA A receptors with bicuculline or by activating metabotropic glutamate receptors with trans -1- aminocyclopentane-1,3-dicarboxylic acid ( trans -ACPD). 3. A second form of synchronous oscillation was activated by acute application of kainic acid (10 [mu]M), had dominant frequencies of 1-5 Hz, and was reversibly abolished by DNQX. Low concentrations of domoic acid mimicked the effects of kainate, but [alpha]-amino-3-hydroxy-5-methyl-4-isoxazole-propionic acid (AMPA) or quisqualic acid did not, suggesting a role for the GluR5-7 and KA1-2 glutamate receptor subunits. 4. Surgical isolation of cortical layers showed that spontaneous NMDA receptor-dependent oscillations originated within layer 5 exclusively, but kainate receptor-dependent oscillations were uniquely generated by neurons in layers 2/3. 5. Our results suggest that neocortical neurons in layers 2/3 and layer 5 can independently generate two distinct forms of rhythmic population activity, each dependent upon activation of a different subtype of glutamate receptor.

Received 25 September 1995; accepted in final form 6 November 1995.
APS Manuscript Number J637-5.
Article publication pending J. Neurophysiol.
ISSN 1080-4757 Copyright 1995 The American Physiological Society.
Published in APStracts on 30 November 95