Stimulus and cell-specific expression of c-x-c and c-c chemokines by pulmonary stromal cell populations. Lukacs, N. W., S. L. Kunkel, R. Allen, H. L. Evanoff, C. L. Shaklee, J. S. Sherman, M. D. Burdick, and R. M. Strieter. University of Michigan Medical School, Dept. of Pathology and Internal Medicine; Div of Pulmonary and Critical Care, Ann Arbor, MI.
APStracts 2:0020L, 1995.
Chronic inflammatory responses in the lung rely on the continual recruitment of leukocytes to the site of inflammation. Recent data has demonstrated a possible role for stromal cell-derived chemokines in leukocyte recruitment. In the present study we examined the production of IL-8 and ENA-78, members of the C-X-C family of chemokines, and MIP-1 a and b, members of the C-C chemokine family, from pulmonary smooth muscle and endothelial cells. The production of IL-8 and ENA-78 was induced by early response cytokines, IL-1 and TNF, but not by immune-associated cytokines, IL-4, IL-10, or IFN-g. In contrast, the production of MIP-1 a/b by pulmonary vascular smooth muscle cells increased when stimulated by immune-associated cytokines, as well as with IL-1b and TNF. The level of MIP-1 a production induced by immune-associated cytokines, IL-4, IFN-g and IL-10. The production of MIP-1 b in response to immune-associated cytokines, IL-4, IFN-g and IL-10. Human pulmonary artery endothelial cells did not generate MIP-1 a or b in response to graded doses of any of the cytokines. These data demonstrate differential induction of C-X-C and C-C chemokines from nonimmune stromal cell populations. The C-X-C chemokines are induced primarily by early response cytokines, IL-1b and TNF. However, the C-C chemokines were induced in smooth muscle cells, but not endothelial cells, by both early response and immune-associated cytokine stimulation. These mechanisms may be pertinent in the development and regulation of acute and chronic inflammatory responses.

Received 1 August 1994; accepted in final form 27 December 1994.
APS Manuscript Number L217-4.
Article publication pending Am. J. Physiol. (Lung Cell. Mol.
Physiology).
ISSN 1080-4757 Copyright 1995 The American Physiological Society.
Published in APStracts on 24 February 1995.