Cl- channels in basolateral renal medullary vesicles ix. comparison of channels from excised patches of cultured mouse mtal cells and basolateral medullary vesicles. Reeves, W. Brian, Christopher J. Winters, Dragana M. Filipovic, Thomas E. Andreoli. Division of Nephrology, Department of Internal Medicine, University of Arkansas College of Medicine and John L. McClellan Veterans Administration Hospital, Little Rock, Arkansas
APStracts 2:0089F, 1995.
The experiments in this paper compared Cl- channels fused into bilayers from rabbit outer medullary vesicles with Cl- channels in excised patches of basolateral membranes from cultured mouse mTAL cells; and evaluated whether the latter were plausible candidates for the Cl- channels mediating net NaCl absorption in microperfused mouse mTAL segments. The unique signature characteristics of Cl- channels incorporated into lipid bilayers from outer medullary vesicles include: activation of Po by increases in the Cl- concentrations bathing intracellular faces; activation of Po by (PKA + ATP) when the Cl- concentrations bathing intracellular faces are low; and no effect of (PKA + ATP) on Po with high cytoplasmic face Cl- concentrations. These same properties were observed in Cl- channels studied using excised patches of basolateral membranes from mouse mTAL cells. Moreover, both in bilayers and in excised patches, the sharpest fractional increase in Cl- channel Po occurred with cytosolic face Cl- concentration increases to values similar to the ADH-dependent values of intracellular Cl- activity in microperfused mouse mTAL segments. And these fractional Po increases were adequate to account quantitatively for the ADH-dependent increase in basolateral membrane Cl- conductance in microperfused mouse mTAL segments. Thus the excised patch basolateral Cl- channels reported in this paper are reasonable candidates for those mediating net Cl- absorption in the mTAL.

Received 22 September 1994; accepted in final form 3 April 1995.
APS Manuscript Number F340-4.
Article publication pending Am. J. Physiol. (Renal Fluid Electrolyte
Physiology).
ISSN 1080-4757 Copyright 1995 The American Physiological Society.
Published in APStracts on 30 May 1995.