Effects of transmural field stimulation in isolated smooth muscle of the human rectum and internal anal sphincter . Glavind, E. B., A. Forman, G. Madsen, A. T[stod]ottrup. Dept. of Obstetrics and Gynecology, University Hospital of Aarhus and Surgical Research Unit, Dept. of Surgery L, section Amtssygehuset, University Hospital of Aarhus, Denmark
APStracts 3:0236G, 1996.
Smooth muscle preparations representing the circular muscle layer of the most distal rectum, and proximal and distal human internal anal sphincter (IAS) mounted in organ baths for recording of isometric tension developed spontaneous tension. Transmural electrical field stimulation (TMS) induced frequency- and impulse duration- dependent relaxations, sensitive to tetrodotoxin in the stimulation range 0.5 - 40 Hz and 0.04 - 0.6 ms. Poststimulus contractions were most frequent and prominent in rectal preparations. Maximal relaxations were comparable in the three locations and achieved at 10 Hz and 0.4 ms. The frequency inducing half maximal response (F50 ) was lower in rectal strips compared to IAS. Phentolamine (10-6 M) enhanced relaxations and diminished off-contractions at 40 Hz in distal IAS. Nw-nitro-L-arginine (L-NNA) concentration-dependently inhibited both relaxations and off-contractions (10 Hz, 0.4 ms). pD2 values of L-NNA were lower in rectal muscle compared to IAS. L-arginine (10-4 M) inhibited the blocking effect of L-NNA. In half of the preparations L-NNA reversed the relaxations to duration-contractions (15 - 40 Hz), which were inhibited by atropine in rectal preparations and phentolamine in IAS. In conclusion, the excitatory innervation of the IAS is [alpha]-adrenergic and cholinergic in the rectum. A product of the L-arginine-NO pathway mediates the TMS-induced inhibition of the muscle and is also involved in poststimulus contractions.

Received 30 January 1996; accepted in final form 6 November 1996.
APS Manuscript Number G41-6.
Article publication pending Am. J. Physiol. (Gastrointest. Liver
Physiology).
ISSN 1080-4757 Copyright 1996 The American Physiological Society.
Published in APStracts on 13 November 1996