Chronic lung injury in preterm lambs: disordered pulmonary elastin deposition. Pierce, Richard A, Kurt H Albertine, Barry C Starcher, John F Bohnsack, David P Carlton, and Richard D Bland. Department of Internal Medicine, Washington University School of Medicine at Barnes-Jewish Hospital, St. Louis, MO 63110, Department of Pediatrics, Division of Developmental Lung Biology, University of Utah School of Medicine, Salt Lake City, UT 84132, Department of Biochemistry, University of Texas, Tyler, TX 75710
APStracts 3:0209L, 1996.
Prolonged mechanical ventilation of premature neonates is often associated with abnormal morphological development of the lung and chronic lung disease, sometimes called bronchopulmonary dysplasia (BPD). Impaired alveolar development is a hallmark of this disease. To better understand the effects of mechanical ventilation on lung elastin expression, we studied lung tissue from 10 preterm lambs (gestation = 125 d; term =148 d) mechanically ventilated for 3-4 weeks at a respirator rate of 20 breaths/min and tidal volume of 15+/-5 ml/kg (n=5) or 60 breaths/min and tidal volume of 5+/-2 ml/kg (n=5). Histopathology showed increased elastin accumulation and abnormal morphological development in the ventilated groups. Postmortem lung desmosine content was increased significantly in the 20 breaths/min group. Tropoelastin mRNA expression was increased in both ventilated groups. In situ hybridization localized increased tropoelastin mRNA expression to sites of accumulated elastin in extended alveolar walls with scant, attenuated secondary crests. Lung collagen content as assessed by the amount of hydroxyproline in lung tissue, was similar to controls. These data suggest that excessive production and accumulation of elastin is associated with chronic lung injury from prolonged mechanical ventilation after premature birth.

Received 21 May 1996; accepted in final form 15 October 1996.
APS Manuscript Number L145-6.
Article publication pending Am. J. Physiol. (Lung Cell. Mol.
Physiology).
ISSN 1080-4757 Copyright 1996 The American Physiological Society.
Published in APStracts on 31 December 1996