Photoperiod-dependent correlation between light-induced scn c-fos expression and resetting of circadian phase. Tr[acute]avn[acute]ickov[acute]a, Zdena, Alena Sumov[acute]a, Robin Peters, William J. Schwartz, Helena Illnerov[acute]a. Institute of Physiology, Academy of Sciences of the Czech Republic, 142 20 Prague 4, Czech Republic, Department of Neurology, University of Massachusetts Medical School, Worcester, MA 01655 U.S.A.
APStracts 3:0146R, 1996.
In rodents, brief light pulses that shift the phase of the circadian locomotor rhythm also increase c-fos gene expression in the suprachiasmatic nucleus (SCN), site of an endogenous clock that regulates such rhythmicity. Since the magnitude of photic phase shifts varies when light pulses are applied at different time points over the course of the subjective night, we examined the degree of SCN c-fos gene expression after single 30-min light pulses were delivered at time points that spanned the early and late subjective night in rats maintained in either short (LD 8:16) or long (LD 16:8) photoperiods. The light- induced level of c-fos mRNA and the number of cells expressing immunoreactive c-Fos protein were measured in the SCN by in situ hybridization and immunohistochemistry, respectively, and compared to the magnitude of the corresponding phase shifts of the circadian rhythm of pineal N- acetyltransferase (NAT) activity. We found a robust correlation between c-fos photoinduction and NAT phase shifts, but this relationship was dependent on photoperiod. The degree of c-fos gene expression was strongly correlated with the magnitude of NAT phase advances and delays under the short photoperiod, and with phase advances under the long photoperiod, but not with phase delays under the long photoperiod. The data suggest that c-fos gene expression in the SCN may be involved in the photic resetting of the pineal NAT rhythm. Under the long photoperiod, however, the magnitude of phase delays may be limited by the functional state of the circadian pacemaking system.

Received 3 November 1995; accepted in final form 13 March 1996.
APS Manuscript Number R686-5.
Article publication pending Am. J. Physiol. (Regulatory Integrative
Comp. Physiology).
ISSN 1080-4757 Copyright 1996 The American Physiological Society.
Published in APStracts on 23 April 96