Inhibition of adenosine1 receptor-mediated preglomerular vasoconstriction in at1areceptor deficient mice. Traynor, Timothy, Tianxin Yang, Yuning G. Huang, Lois Arend, Michael I. Oliverio, Thomas Coffman, Josie P. Briggs, and J[umlaut]urgen Schnermann. 1Departments of Physiology and Internal Medicine, University of Michigan, Ann Arbor, Michigan, 2DKUHD, NIDDK, NIH, Bethesda, Maryland, and 3Department of Medicine, Duke University, Durham, North Carolina
APStracts 5:0153F, 1998.
The effect of the adenosine1 receptor agonist cyclohexyladenosine (CHA) on glomerular vascular reactivity was studied in male angiotensin II 1A (AT1A) receptor knockout mice (9). Vascular reactivity was assessed as the response of stop flow pressure (PSF) to infusion of CHA into loops of Henle using micropuncture techniques. In AT1A +/+ mice at ambient arterial blood pressure (96.7 +/- 2.8 mm Hg), the presence of CHA (10 -5 M) in the perfusate increased PSF responses from 6.8 +/- 0.6 to 14.3 +/- 0.9 mm Hg when the loop of Henle of the index nephron was perfused, and from 0.7 +/- 0.3 to 12.3 +/- 1.0 mm Hg when the loop of an adjacent nephron was perfused. At reduced arterial blood pressure (82.8 +/- 1.3 mm Hg) index nephron perfusion with CHA increased PSF responses from 4.5 +/- 0.3 mm Hg to 9.4 +/- 0.4 mm Hg. In AT1A -/- mice with a mean arterial blood pressure of 80 +/- 1.9 mm Hg, CHA increased PSF responses only from 0.1 +/- 0.3 to 3.6 +/- 0.54 mm Hg during index nephron perfusion and from 0.25 +/- 0.2 to 2.7 +/- 0.55 mm Hg during adjacent nephron perfusion, significantly less than in wild type animals (p<0.001). Responses to CHA were intermediate in AT1A +/- mice. Thus, AT1A receptor knockout mice show a markedly reduced constrictor response to CHA both in the presence and absence of simultaneous activation of the TGF system. These data support the notion of a functional interaction between adenosine and angiotensin II in the regulation of afferent arteriolar tone.

Received 13 April 1998; accepted in final form 27 March 1998.
APS Manuscript Number F83-8.
Article publication pending Am. J. Physiol. (Renal Physiology).
ISSN 1080-4757 Copyright 1998 The American Physiological Society.
Published in APStracts on 21 September 1998