Memory and the Brain: Neuroanatomical and Functional Imaging Correlates of Memory and Memory Disorders

Hans Markowitsch

University of Bielefeld


Prerequisites: Read at least on my chapters on memory and amnesia given below.


 Course Description:

Memory nowadays is divided according to time and contents.  Along the dimension time it is distinguished between short-term or on-line memory (“working memory”) and long-term memory, with short-term memory lasting from seconds to minutes and long-term memory referring to everything of longer duration. With respect to contents, there are three main directions: A few people consider memory to be a unitary system. Most scientists divide memory into several systems. A second approach is associated with the name of Larry Squire (“declarative and non-declarative forms of memory”), and the third with that of Endel Tulving (“episodic and semantic memory systems as well as 3 other forms”). Tulving and Markowitsch at present divide memory into five systems and suggest different anatomical networks for each of these systems. Furthermore, they consider it possible to arrange them hierarchically. The most complex memory system is the episodic-autobiographical one which requires self-conscious reflection and is embedded in the dimensions of time and locus. It frequently is affect-related and allows mental time traveling. Semantic memory, on the other hand, is context-free fact memory. On the more automatic, implicit level, perceptual memory, procedural memory and the priming system constitute the other three long-term memory systems with perceptual memory allowing the assessment of novelty/familiarity of perceptual stimuli, procedural memory referring largely to sensory-motor skills and simple conditioning and priming to an enhanced identification of objects.
On the brain level, working memory recruits prefrontal and (left) parietal structures. For information acquisition, episodic and – though to a much lesser degree – semantic memory engage limbic structures and portions of the prefrontal cortex, while perceptual memory and priming are principally a matter of poly- and unimodal neocortical regions, and procedural memory one of the basal ganglia, possibly of cortical premotor regions and cerebellar structures. For the last three memory systems, information storage and information retrieval engage the same structures necessary for acquisition or encoding, while for the episodic and memory systems the situation is more complex. Both of these are represented in neocortical nets, the episodic memory system furthermore possibly recruits limbic structures. There seems to be a preponderance of right-hemispheric nets for episodic and of left-hemispheric ones for semantic memories. Similarly, episodic information retrieval is triggered via right-hemispheric prefrontal and anterior structures, while semantic memory retrieval activates the same net of the left hemisphere.
These networks for information processing have been established on the basis of both patient data and data in normal subjects using functional imaging methods. Our own research centers on studying patients with focal neurological damage who have major forms of episodic anterograde and/or retrograde amnesia, on studying the processing of autobiographical memory in normals subjects with positron emission tomography and functional magnetic resonance imaging. Recent interesting features of this work include differing brain networks engaged in old versus more recent autobiographical memories, different anatomical representations of memories with a positive (“happy”) and negative (“sad”) emotional background. Furthermore, our recent research demonstrates that in part similar brain regions appear metabolically suppressed in patients with socalled non-organic, that is, psychiatric or psychogenic amnesias. For these, an increased release of stress hormones is seen as basis for what can be termed a mnestic block syndrome. It will be discussed whether the results of modern functional imaging methods allow to bridge the gap between patients with manifest organic brain tissue damage and patients with “functional” amnesia induced by psychic stress or trauma conditions.



Course Reading List


     Markowitsch, H.J. (2000). Memory and amnesia. In M.-M. Mesulam (Ed.), Principles of cognitive and behavioral neurology (pp. 257-293). New York: Oxford University Press.


Markowitsch, H.J. (2000). Neuroanatomy of memory. In E. Tulving & F.I.M. Craik (Eds.), The Oxford handbook of memory (pp. 465-484). New York: Oxford University Press.


Markowitsch, H.J. (2000). Anatomical bases of memory disorders. In M.S. Gazzaniga (Ed.), The new cognitive neurosciences (2nd ed.). (pp. 781-795). Cambridge, MA: MIT Press.


Markowitsch, H.J. (2003). Autonoëtic consciousness. In A.S. David & T. Kircher (Eds.), The self and schizophrenia: A neuropsychological perspective (in press). Cambridge: Cambridge University Press.

Fink, G.R., Markowitsch, H.J., Reinkemeier, M., Bruckbauer, T., Kessler, J. & Heiss, W.-D. (1996). Cerebral representation of one’s own past: neural networks involved in autobiographical memory. Journal of Neuroscience, 16, 4275-4282.


Markowitsch, H.J. (1996). Organic and psychogenic retrograde amnesia: two sides of the same coin? Neurocase, 2, 357-371.


Markowitsch, H.J. (1999). Functional neuroimaging correlates of functional amnesia. Memory, 7, 561-583.


Markowitsch, H.J., Kessler, J., Weber-Luxenburger, G. , Van der Ven, C. & Heiss, W.-D. (2000). Neuroimaging and behavioral correlates of recovery from ‘mnestic block syndrome’ and other cognitive deteriorations. Neuropsychiatry, Neuropsychology, and Behavioral Neurology, 13, 60-66.


Markowitsch, H.J. (2002). Functional retrograde amnesia – mnestic block syndrome. Cortex, 38, 651-654.


Piefke, M., Weiss, P.H., Zilles, K., Markowitsch, H.J. & Fink, G.R. (2003). Differential remoteness and emotional tone modulate the neural correlates of autobiographical memory. Brain, in press.